OP-BRAI190181 1..15 Vector-mediated L-3,4-dihydroxyphenylalanine delivery reverses motor impairments in a primate model of Parkinson’s disease Carl Rosenblad,1,2 Qin Li,3 Elsa Y. Pioli,3 Sandra Dovero,4,5 André SLM Antunes,6 Leticia Agúndez,6 Martino Bardelli,6 R. Michael Linden,6 Els Henckaerts,6Anders Björklund,2 Erwan Bezard3,4,5 and Tomas Björklund2,7 Ever since its introduction 40 years a

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awp328 482..495 BRAIN A JOURNAL OF NEUROLOGY The A9 dopamine neuron component in grafts of ventral mesencephalon is an important determinant for recovery of motor function in a rat model of Parkinson’s disease Shane Grealish,1 Marie E. Jönsson,1 Meng Li,2 Deniz Kirik,3 Anders Björklund1 and Lachlan H. Thompson1,4 1 Division of Neurobiology, Wallenberg Neuroscience Centre, Lund University, Lund,

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Neuropeptide Y and its role in CNS disease and repair Experimental Neurology 238 (2012) 265–272 Contents lists available at SciVerse ScienceDirect Experimental Neurology j ourna l homepage: www.e lsev ie r .com/ locate /yexnr Review Neuropeptide Y and its role in CNS disease and repair M. Decressac a,⁎, R.A. Barker b a Wallenberg Neuroscience Center, Department of Experimental Medical Sciences, Lu

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Cardoso_et_al-2018-Journal_of_Comparative_Neurology(1).pdf R E S E A R CH AR T I C L E Target-specific forebrain projections and appropriate synaptic inputs of hESC-derived dopamine neurons grafted to the midbrain of parkinsonian rats Tiago Cardoso1,2 | Andrew F. Adler1,2 | Bengt Mattsson1,2 | Deirdre B. Hoban1,2 | Sara Nolbrant1,2 | Jenny Nelander Wahlestedt1,2 | Agnete Kirkeby1,3 | Shane Grealis

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Journal of Parkinson’s Disease 6 (2016) 57–63 DOI 10.3233/JPD-160798 IOS Press 57 Commentary Are Stem Cell-Based Therapies for Parkinson’s Disease Ready for the Clinic in 2016? Roger A. Barkera,b,∗, Malin Parmarb, Agnete Kirkebyb, Anders Björklundb, Lachlan Thompsonc and Patrik Brundind aJohn van Geest Centre for Brain Repair, Department of Clinical Neurosciences, University of Cambridge, Forvie

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Journal of Parkinson’s Disease 7 (2017) S23–S33 DOI 10.3233/JPD-179002 IOS Press S23 Review Replacing Dopamine Neurons in Parkinson’s Disease: How did it happen? Anders Björklunda,∗ and Olle Lindvallb,∗ aDepartment of Experimental Medical Science, Wallenberg Neuroscience Center, Lund, Sweden bDepartment of Clinical Sciences, and Lund Stem Cell Center, Division of Neurology, University Hospital, L

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Invoice VAT NO: NL858213928B01 BioPartner 4, Robert Boyleweg 4, 2333 CG, Leiden, Netherlands GenScript Biotech (Netherlands) B.V. Customer NO: G0146407P0 Order NO: U8027FG070 Invoice NO: 92364337 Invoice Date: 2020.07.21 Quote NO: BILL TO SHIP TO Jenny G Johansson Lunds Universitet Box188, Lund, -, 221 00,SE - VAT No: Lunds Universitet Jenny Johansson BMC Godsmottagning D09, Lund, -, 22241,SE 46 4

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U nc or re ct ed A ut ho r P ro of Journal of Parkinson’s Disease xx (20xx) x–xx DOI 10.3233/JPD-202061 IOS Press 1 Research Report1 BDNF Overexpression Increases Striatal D3 Receptor Level at Striatal Neurons and Exacerbates D1-Receptor Agonist-Induced Dyskinesia 2 3 4 5 Simona Scheggia,1, Francesca Rossib,1, Sara Corsib, Silvia Fannib, Elisabetta Troncib, Ludovica Congiub, Romina Vargiub, Carla

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© 20 13 L an de s B io sc ie nc e. D o no t d is tri bu te . www.landesbioscience.com Autophagy 1 Autophagy 9:8, 1–3; August 2013; © 2013 Landes Bioscience AutophAgic punctum AutophAgic punctum Punctum to: Decressac M, Mattsson B, Weikop P, Lundblad M, Jakobsson J, Björklund A. TFEB-mediated autophagy rescues midbrain dopamine neurons from α-synuclein toxicity. Proc Natl Acad Sci U S A 2013; 110:E

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JW-MDS#130009 1..9 Study of the Antidyskinetic Effect of Eltoprazine in Animal Models of Levodopa-Induced Dyskinesia Erwan Bezard, PhD,1,2,3 Elisabetta Tronci, PhD,4,5 Elsa Y. Pioli, PhD,6 Qin Li, PhD,3,6 Gregory Porras, PhD,6 Anders Bj€orklund, MD,5 and Manolo Carta, PhD4,5* 1Institute for Neurodegenerative Diseases, Bordeaux University, Bordeaux, France 2Institute for Neurodegenerative Diseases,

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Modeling Parkinson’s disease pathology by combination of fibril seeds and α-synuclein overexpression in the rat brain Modeling Parkinson’s disease pathology by combination of fibril seeds and α-synuclein overexpression in the rat brain Poonam Thakura,1,2, Ludivine S. Bregera,1, Martin Lundblada, Oi Wan Wana, Bengt Mattssona, Kelvin C. Lukb,c, Virginia M. Y. Leeb,c, John Q. Trojanowskib,c, and Ande

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pnas201200575 1..7 Impaired neurotransmission caused by overexpression of α-synuclein in nigral dopamine neurons Martin Lundblad1, Mickael Decressac, Bengt Mattsson, and Anders Björklund1 Wallenberg Neuroscience Center, Department of Experimental Medical Sciences, Lund University, Lund 221 84, Sweden This contribution is part of the special series of Inaugural Articles by members of the National A

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stm0420Editor_BjorkF.indd E D I T O R I A L www.ScienceTranslationalMedicine.org 20 April 2011 Vol 3 Issue 79 79ed1 1 C L I N I C A L T R I A L S NEUROLOGICAL DISEASES ARE AMONG THE MOST INTERESTING APPLICATIONS FOR gene therapy. A er decades of preclinical development, viral vector–based gene transfer has now reached the clinic, and early-stage clinical trials are currently in progress in three a

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