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Schedule and course documents

On this page you will find your course schedule, literature list and syllabus. Click on the course name and the documents will be visible through links. Make sure the document you open concerns the intended course. Hyperlinks are added to the page manually and mistakes can occur. Contact webbredaktor [at] soc [dot] lu [dot] se (webbredaktor[at]soc[dot]lu[dot]se) if you find deviations. Relevant li

https://www.gender.lu.se/education/student-information/schedule-and-course-documents - 2025-07-27

Visiting scholars

The Department of Gender Studies have had the pleasure of hosting various guest researchers through the years. Here we list researchers who have been at the department between 2015 and now. Illustration: Sarah Hirani Current visiting scholars Lærke Munk Rigtrup-Lindemann, Doctoral student, Nord University in Norwegian Sápmi. Lærke Munk Rigtrup-Lindemann is PhD-student in sociology from Nord Univer

https://www.gender.lu.se/research-0/visiting-scholars - 2025-07-27

Research seminars

The gender studies division's research seminars with international and national researchers. On this page you will find information about current seminar series. Every semester, the Division of Gender Studies host research seminars where researchers from around the world are invited to present and discuss their research. Our research seminars are open to the public and usually held in English. Gen

https://www.gender.lu.se/research-0/research-seminars - 2025-07-27

Gender studies: Feminist Methodologies and Methods (7.5 credits)

GNVN33 SPRING This course offers a gender studies analysis of method and methodology as an intellectual framework within the social sciences. This involves to understand and critical reflect on current feminist theories on epistemology, methodology and method within gender studies. The course aims to provide the student with theoretical and practical knowledge on methods for research design, data

https://www.gender.lu.se/education/graduate-courses/gender-studies-feminist-methodologies-and-methods-75-credits - 2025-07-27

Gender Studies: Gender in a Global World (7.5 credits)

GNVN05 FALL Would you like to develop your knowledge in gender studies at the advanced level? This course offers you an opportunity to deepen your understanding of intersectional perspectives on relations between gender, on the one hand, and migration, globalization, power and change, on the other. The course engages with current events and you will carry out exercises in feminist analysis of the

https://www.gender.lu.se/education/graduate-courses/gender-studies-gender-global-world-75-credits - 2025-07-27

Gender Studies: Gender and Nation in Europe (7.5 credits)

GNVN06 FALL The course focuses on the economic, political and cultural changes in gender regimes and conviviality in Europe. It provides students with the opportunity to specialise in theories and analyses of how gender, migration, sexuality and family have become central phenomena in several complex and contradictory ways in the contemporary political development in Europe, focusing on austerity

https://www.gender.lu.se/education/graduate-courses/gender-studies-gender-and-nation-europe-75-credits - 2025-07-27

Student information

Welcome to the Division of Gender Studies On this page we have collected information to all admitted students at the Division of Gender Studies. Schedule, reading list and syllabus Find your course schedule and course material for the semester. AdmissionWelcome lettersNew students: ChecklistCanvas: Learning platformStudent support at Lund UniversityGeneral questions about your application and admi

https://www.gender.lu.se/education/student-information - 2025-07-27

Early postnatal behavioral, cellular, and molecular changes in models of Huntington disease are reversible by HDAC inhibition

Siebzehnrübl FA, Raber KA, Urbach YK, Schulze-Krebs A, Canneva F, Moceri S, Habermeyer J, Achoui D, Gupta B, Steindler DA, Stephan M, Nguyen HP, Bonin M, Riess O, Bauer A, Aigner L, Couillard-Despres S, Paucar MA, Svenningsson P, Osmand A, Andreew A, Zabel C, Weiss A, Kuhn R, Moussaoui S, Blockx I, Van der Linden A, Cheong RY, Roybon L, Petersén Å and von Hörsten S.PNAS 115 (37) E8765-E8774 (2018)

https://www.huntington-research.lu.se/early-postnatal-behavioral-cellular-and-molecular-changes-models-huntington-disease-are-reversible - 2025-07-27

Telomere dysfunction triggers extensive DNA fragmentation and evolution of complex chromosome abnormalities in human malignant tumors

Gisselsson D, Jonson T, Petersén Å, Strombeck B, Dal Cin P, Hoglund M, Mitelman F, Mertens F and Mandahl N.Proceedings of the National Academy of Sciences USA 98: 12683-12688 (2001)AbstractAlthough mechanisms for chromosomal instability in tumors have been described in animal and in vitro models, little is known about these processes in man. To explore cytogenetic evolution in human tumors, chromo

https://www.huntington-research.lu.se/telomere-dysfunction-triggers-extensive-dna-fragmentation-and-evolution-complex-chromosome - 2025-07-27

Huntingtons sjukdom - ännu ett galet protein?

Petersén Å, Hansson O and Brundin P.In Swedish. Läkartidningen 50: 5756-5761 (2001)AbstractHuntingtons sjukdom är en av flera kända neurologiska sjukdomar som beror på en expanderad CAG-repetition. Symtomen karakteriseras av motoriska, psykiska och kognitiva störningar. Muterat huntingtin uttrycks i hela kroppen, och neurodegeneration och aggregering av det muterade proteinet huntingtin sker i bas

https://www.huntington-research.lu.se/huntingtons-sjukdom-annu-ett-galet-protein - 2025-07-27

Oxidative stress, mitochondrial permeability transition and activation of caspases in calcium ionophore A23187-induced death of cultured striatal neurons

Petersén Å, Castilho RF, Hansson O, Wieloch T and Brundin P.Brain Research 857: 20-29 (2000)AbstractDisruption of intracellular calcium homeostasis is thought to play a role in neurodegenerative disorders such as Huntington's disease (HD). To study different aspects of putative pathogenic mechanisms in HD, we aimed to establish an in vitro model of calcium-induced toxicity in striatal neurons. The

https://www.huntington-research.lu.se/oxidative-stress-mitochondrial-permeability-transition-and-activation-caspases-calcium-ionophore - 2025-07-27

Caspase inhibition increases embryonic striatal graft survival

Mundt-Petersén U, Petersén Å, Emgård M, Dunnett SB and Brundin P.Experimental Neurology 164:112-120 (2000)AbstractIn transplants of embryonic striatal cells placed into the excitotoxically lesioned rat striatum (a model of Huntington's disease), as many as 60 to 90% of the grafted cells are believed to die. Caspase activation is part of a cascade of events that can lead to apoptosis. We investigat

https://www.huntington-research.lu.se/caspase-inhibition-increases-embryonic-striatal-graft-survival - 2025-07-27

Grafting of nigral tissue hibernated with tirilazad mesylate and glial cell-line derived neurotrophic factor

Petersén Å, Hansson O, Emgård M and Brundin P.Cell Transplantation 9: 577-584 (2000)AbstractTransplantation of embryonic ventral mesencephalon is a potential therapy for patients with Parkinson's disease. As only around 5-10% of embryonic dopaminergic neurons survive grafting into the adult striatum, it is considered necessary to use multiple donor embryos. To increase the survival of the grafted

https://www.huntington-research.lu.se/grafting-nigral-tissue-hibernated-tirilazad-mesylate-and-glial-cell-line-derived-neurotrophic-factor - 2025-07-27

Improving the survival of grafted dopaminergic neurons: a review over current approaches

Brundin P, Karlsson J, Emgård M, Kaminski Schierle GS, Hansson O, Petersén Å and Castilho RF.Cell Transplantation 9: 179-195 (2000)AbstractNeural transplantation is developing into a therapeutic alternative in Parkinson's disease. A major limiting factor is that only 3-20% of grafted dopamine neurons survive the procedure. Recent advances regarding how and when the neurons die indicate that events

https://www.huntington-research.lu.se/improving-survival-grafted-dopaminergic-neurons-review-over-current-approaches - 2025-07-27

Transgenic mice expressing a Huntington's disease mutation are resistant to quinolinic acid-induced striatal excitotoxicity

Hansson O, Petersén Å, Leist M, Nicotera P, Castilho RF and Brundin P.Proceedings of the National Academy of Sciences USA 96: 8727-8732 (1999)AbstractHuntington's disease (HD) is a hereditary neurodegenerative disorder presenting with chorea, dementia, and extensive striatal neuronal death. The mechanism through which the widely expressed mutant HD gene mediates a slowly progressing striatal neuro

https://www.huntington-research.lu.se/transgenic-mice-expressing-huntingtons-disease-mutation-are-resistant-quinolinic-acid-induced - 2025-07-27

Impact of a preceding excitotoxic lesion and treatment with ciliary neurotrophic factor on striatal graft survival

Petersén Å, Emgård M and Brundin P.Brain Research Bulletin 50: 275-281 (1999)AbstractThe survival of grafted embryonic striatal tissue, dissected from the lateral ganglionic eminence, depends on the status of the host striatum. We found significantly larger volumes of surviving graft tissue and of striatal-like tissue (P-zone) within the graft, when the host striatum had been subjected to an excit

https://www.huntington-research.lu.se/impact-preceding-excitotoxic-lesion-and-treatment-ciliary-neurotrophic-factor-striatal-graft - 2025-07-27

Effect of CNTF and calcium-ionophore A23187-induced death in cultured embryonic stratal neurons

Petersén Å and Brundin P.Experimental Neurology 160: 402-412 (1999)AbstractCiliary neurotrophic factor (CNTF) has a protective effect on the striatum in animal models of Huntington's disease. However, the mechanism through which it exerts its effect is not clear. In this study, we show that there is a concentration-dependent direct protective effect of CNTF against N-methyl-D-aspartate-mediated ex

https://www.huntington-research.lu.se/effect-cntf-and-calcium-ionophore-a23187-induced-death-cultured-embryonic-stratal-neurons - 2025-07-27

Recent advances on the pathogenesis of Huntington's disease

Petersén Å, Mani K and Brundin P.Experimental Neurology 157: 1-18 (1999)AbstractWe review recent advances regarding the pathogenesis of Huntington's disease (HD). This genetic neurodegenerative disorder is caused by an expanded CAG repeat in a gene coding for a protein, with unknown function, called huntingtin. There is selective death of striatal and cortical neurons. Both in patients and a trans

https://www.huntington-research.lu.se/recent-advances-pathogenesis-huntingtons-disease - 2025-07-27

SIRT1 is increased in affected brain regions and hypothalamic metabolic pathways are altered in Huntington disease

Baldo B, Gabery S, Soylu-Kucharz R, Cheong RY, Henningsen JB, Englund E, McLean C, Kirik D, Halliday G and Petersén Å.Neuropathology and Applied Neurobiology 45 (4): 361-379 (2019)AbstractAIMS:Metabolic dysfunction is involved in modulating the disease process in Huntington disease (HD) but the underlying mechanisms are not known. The aim of this study was to investigate if the metabolic regulator

https://www.huntington-research.lu.se/sirt1-increased-affected-brain-regions-and-hypothalamic-metabolic-pathways-are-altered-huntington - 2025-07-27